Always happy to share! I’m genuinely happy you read what i wrote!

KPD https://en.wikipedia.org/wiki/Ketosis-prone_diabetes ?

You might find this interesting Ketosis, ketoacidosis and very-low-calorie diets: putting the record straight - 2011

TLDR: DKA and Ketosis are distinct biological states, and eating carbohydrates does not provide a benefit in avoiding DKA. i.e. DKA is resolved with insulin and not injecting/consuming glucose.

Update - Above you said your on a SLGT2 inhibitor, which does decrease insulin levels, so there is a DKA risk with that medication by itself, with your KPD condition I agree its probably not great to combine SLGT2i medication and a strict ketogenic diet

3.5.2.2.2 Medications that increase ketoacidosis risk - SGLT2 inhibitors: These medications carry a risk of euglycaemic ketoacidosis. TCR alone cannot cause ketoacidosis, but it may enhance the risk posed by SGLT2i by lowering insulin concentrations because insulin inhibits ketone formation. SGLT2i-induced ketoacidosis may occur with normal BG concentrations, and this heightens the risk of ketoacidosis going unrecognised. It is worth noting that a VLCD (typically less than 50 g of carbohydrate a day) can produce a physiologically normal state of ketosis, that should not be confused with the pathological state of diabetic ketoacidosis. Despite recent literature supporting slight cardiovascular risk reduction and renal protection of SGLT2i, it is recommended that SGLT2i are used with caution in those adhering to a low carbohydrate eating plan. It is appropriate to stop SGLT2i in many cases, particularly in those adhering to a VLCD (30–50 g/day). A GLP-1 agonist is a safer choice as a second-line agent after metformin. See Murray et al. [299] for an excellent review of the physiology of an LCD mimicking many effects of SGLT2i - From Ketogenic : The Science of Therapeutic Carbohydrate Restriction in Human Health